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Дерматовенерология и эстетическая меДицина № 3/2015
49. abbas a.K., lichtman a.H., Pober j.S. Effector
mechanism of T-cell-mediated immune response.
In: Cellular and molecular Immunology. 2nd
edition, W.b. Saunders. 1994.
50. Fong E.P., bay b.H.: Keloids — the sebum
hypothesis revisited // med Hypotheses. 2002.
58; 264–269.
51. yagi K.I., Dafalla a.a., Osman a.a.: Does an
immune reaction to sebum in wounds cause
keloid scars? // br j Plast Surg. 1979. 32; 223–
225.
52. murray j.C., Pollack S.V., Pinnel S.r. Keloids: a
review // j am acad Dermatol. 1981. 4; 461–470.
53. bock O., Schmid-Ott G., malewski P., mrowietz
u. Quality of life of patients with keloid and
hypertrophic scarring // arch Dermatol res.
2006. 297; 433–438.
54. Peacock E.E. jr, madden j.W., Trier W.C.
biologic basis for the treatment of keloids and
hypertrophic scars // South med j. 1970. 63;
755–759.
55. Crockett D.j. Hypertrophic scars // br j Plast
Surg. 1964. 17; 245–253.
56. Curtis a.S., Seehar G.m. The control of cell
division by tension or diffusion. nature. 1978.
274; 52–53.
57. Sussmann m.D. Effect on increased tissue
traction upon tensile strength of cutaneous
incisions in rats // Proc Soc Exp biol med. 1996.
123; 38–41.
58. Calnan j.S., Copenhagen H.j. autotransplantation
of keloid in man // br j Surg. 1967. 54; 330–335.
59. brody G.S., Peng S.T., landel r.F. The etiology
of hyper-trophic scar contracture: another view
// Plast reconstr Surg. 1981. 67; 673–684.
60. Wang Z., Fong K.D., Phan T.T. et al. Increased
transcriptional response to mechanical strain in
keloid fibroblasts due to increased focal adhesion
complex formation // j Cell Physiol. 2006. 206;
510–517.
61. Eckes b., Zigrino P., Kessler D. et al. Fibroblast-
matrix interactions in wound healing and fibrosis
// matrix biol 19; 2000; 325–332.
62. Hynes r.O. Integrins: versality, modulation, and
signaling in cell adhesion // Cell. 1992. 69; 11–25.
63. Szulgit G., rudolph r., Wandel a., Tenenhaus
m., Panos r. and Gardner H. alterations
in fibroblast alpha1beta1 integrin collagen
expression in keloids and hypertrophic scars // j
Invest Dermatol. 2002. 118; 409–415.
64. langholz O., rockel D., mauch C. et al. Collagen
and collagenase gene expression in the three-
dimensional collagen lattices are differentially
regulated by alpha1 beta1 and alpha 2 and beta1
integrins // j Cell biol. 1995. 131; 1903–1915.
65. ravanti l., Heino j., lopez-Otin C., Kahari V.m.
Induction of collagenase-3 (mmP-13) expression
in human skin fibroblasts by three-dimensional
collagen is mediated by p38 mitogenactivated
protein kinase // j biol Chem. 1999. 174; 2446–
2455.
66. babu m., Diegelmann r., Oliver n. Keloid
fibroblasts exhibit an altered response to TGF-
βeta // j Invest Dermatol. 1992. 99; 650–655.
67. ravanti l., Kдhдri V.m. matrix metalloproteinases
in wound repair // Int j. mol med. 2000. 6; 391–
407.
68. lyons r.m., Gentry l.E., Purchio a.F., moses
H.l. mechanisms of activation of latent
recombinant transforming growth factor beta 1 by
plasmin // j. Cell biol. 1990. 110; 1361–1367.
69. niessen F.b., Spauwen P.H., Schalkwijk j., Kon
m. On the nature of hypertrophic scars and
keloids: a review // Plast reconstr Surg. 1999.
104; 1435–1458.
70. Tredget E.E., nedelec b., Scott P.G., Ghahary
a. Hypertrophic scars, keloids and contractures.
The cellular and molecular basis for therapy
// Surg Clin north am. 1997. 77; 701–730.
71. Wall S.j., bevan D., Thomas D.W. et al.
Differential expression of matrix metalloproteina-
ses during impaired wound healing of the diabetic
mouse // j Invest Dermatol. 2002. 119; 91–98.
72. Sadick H., Herberger a., riedel K. et al. TGF-
βeta1 antisense therapy modulates xpression
of matrix metalloproteinases in keloidderived
fibroblasts // Int j mol med. 2008. 22; 55–60.
73. neely a.n., Clendening C.E., Gardener j.,
Greenhalgh D.G.,Warden G.D. Gelatinase
activity in keloids and hypertrophic scars
// Wound repair regen. 1999. 7; 166–171.
74. Fujiwara m., muragaki y., Ooshima a. Keloid-
derived fibroblasts show increased secretion of
factors involved in collagen turnover and depend
on matrix metalloproteinase for migration // br j
Dermatol, 2005. 153; 295–200.
75. border W.a., noble n.a. Transforming growth
factor beta in tissue fibrosis // n Engl j med.
1994. 331; 1286–1292.
76. Peltonen j., Hsiao l.l., jaakkola S. et al.
activation of collagen gene expression in keloids:
Co-localization of type I and VI collagen and
transforming growth factor beta 1 mrna // j
Invest Dermatol. 1991. 97; 240–248.
77. russell S.b., Trupin K.m., rodriguez E.S.,
russell j.D., Trupin j.S. reduced growth factor
requirement of keloid-derived fibroblasts may
account for tumor growth // Proc natl acad Sci
uSa. 1988. 85; 587–591.
78. Shah m., Foreman D.m., Ferguson m.W. Control
of scarring in adult wounds by neutralising
antibody to transforming growth factor beta
// lancet. 1992. 339; 213–214.
79. Shah m., Foreman D.m., Ferguson m.W.
neutralisation of TGF-βeta 1 and TGF-βeta 2 or
exogenous addition of TGF-βeta 3 to cutaneous
rat wounds reduces scarring // j Cell Sci. 1995.
108; 985–1002.
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